人們一直擔(dān)心,,兒童經(jīng)常使用手機可能引發(fā)腦瘤。但瑞士研究人員說,,他們最新完成的一項有關(guān)研究沒有發(fā)現(xiàn)手機會導(dǎo)致兒童患腦瘤的證據(jù)。
瑞士熱帶與公共衛(wèi)生研究所的研究人員在美國新一期《全國癌癥研究所期刊》上報告說,他們收集了丹麥,、挪威、瑞典和瑞士等國352名7歲至19歲的腦瘤患者使用手機的情況,,并把有關(guān)數(shù)據(jù)與646名同樣年齡段健康孩子的情況進行了對比,。
結(jié)果顯示,,患有腦瘤的兒童并不比沒有患腦瘤的兒童更多地使用手機。研究人員據(jù)此認(rèn)為,,使用手機可能并不是引發(fā)兒童腦瘤的主要原因,。
研究人員建議,如果家長仍然擔(dān)心使用手機會對孩子有不良影響,,不妨讓孩子使用低輻射的手機,,或讓他們在通話時使用耳機等。(生物谷 Bioon.com)
doi:10.1093/jnci/djr244
PMC:
PMID:
Mobile Phone Use and Brain Tumors in Children and Adolescents: A Multicenter Case–Control Study
Denis Aydin, Maria Feychting, Joachim Schüz, Tore Tynes, Tina Veje Andersen, Lisbeth Samsø Schmidt, Aslak Harbo Poulsen, Christoffer Johansen, Michaela Prochazka, Birgitta Lannering, Lars Klæboe, Tone Eggen, Daniela Jenni, Michael Grotzer, Nicolas Von der Weid, Claudia E. Kuehni and Martin Röösli
Many persistent pain states (pain lasting for hours, days, or longer) are poorly treated because of the limitations of existing therapies. Analgesics such as nonsteroidal anti-inflammatory drugs and opioids often provide incomplete pain relief and prolonged use results in the development of severe side effects. Identification of the key mediators of various types of pain could improve such therapies. Here, we tested the hypothesis that hitherto unrecognized cytokines and chemokines might act as mediators in inflammatory pain. We used ultraviolet B (UVB) irradiation to induce persistent, abnormal sensitivity to pain in humans and rats. The expression of more than 90 different inflammatory mediators was measured in treated skin at the peak of UVB-induced hypersensitivity with custom-made