近日,，耶魯醫(yī)學(xué)院的研究者通過(guò)研究表明，孕婦長(zhǎng)期置身于手機(jī)的輻射之中會(huì)影響其后代的大腦發(fā)育,，有可能會(huì)導(dǎo)致嬰兒的大腦過(guò)度活躍,。相關(guān)研究結(jié)果刊登在了國(guó)際雜志Nature旗下雜志Scientific Reports上,。
懷孕期間，長(zhǎng)期暴露于手機(jī)輻射之中會(huì)影響孩子的大腦發(fā)育,，會(huì)引起孩子行為異常(Credit: © Poulsons Photography / Fotolia)
研究者Hugh S Taylor博士表示,，我們的研究首次提供了證據(jù)，嬰兒長(zhǎng)期暴露于手機(jī)輻射下會(huì)影響其成年后的行為,，研究者將手機(jī)放于飼養(yǎng)懷孕的小鼠籠子之上,，確保手機(jī)的輻射作用一直持續(xù)，對(duì)照組為將懷孕小鼠置于無(wú)手機(jī)長(zhǎng)期輻射的環(huán)境中,，當(dāng)這些受輻射的胎兒長(zhǎng)至成年小鼠后,，研究者測(cè)定了其大腦的電動(dòng)生物活性，并且進(jìn)行了一系列的心理和行為能力測(cè)定,，研究者發(fā)現(xiàn)暴露于手機(jī)輻射的小鼠在成年后更趨于亢奮以及記憶力降低,，Taylor將這些行為上的改變歸因于在懷孕時(shí)期胎兒受到輻射使得其大腦前額葉皮質(zhì)區(qū)的神經(jīng)發(fā)育受到影響所致，注意力缺陷伴多動(dòng)障礙（ADHD）就是因?yàn)榇竽X前額葉皮質(zhì)區(qū)發(fā)育障礙所引起的一種疾病,，主要特點(diǎn)也是注意力不集中以及過(guò)渡亢奮,。
研究者Taylor表示，我們目前研究發(fā)現(xiàn)這種類似ADHD的行為問(wèn)題是因?yàn)樽訉m內(nèi)的胎兒受到手機(jī)輻射的影響,，人類中，小孩發(fā)生行為障礙的比例的升高有很大一部分原因是因?yàn)殚L(zhǎng)期暴露于手機(jī)輻射之中,，研究者表示他們后期的研究將會(huì)在人類中進(jìn)行,，以此更好的理解和驗(yàn)證他們的研究發(fā)現(xiàn)，并且給懷孕婦女建立安全的無(wú)輻射環(huán)境,。研究者Aldad說(shuō),，“實(shí)驗(yàn)中的手機(jī)是模擬人類處于輻射的環(huán)境之中的，后期研究將會(huì)使用標(biāo)準(zhǔn)的電磁場(chǎng)輻射生成器來(lái)使得實(shí)驗(yàn)環(huán)境更加精準(zhǔn),。”（生物谷：T.Shen編譯）
doi:10.1038/srep00312
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Fetal Radiofrequency Radiation Exposure From 800-1900 Mhz-Rated Cellular Telephones Affects Neurodevelopment and Behavior in Mice
Tamir S. Aldad,1, 2 Geliang Gan,2 Xiao-Bing Gao2, 3 & Hugh S. Taylor1, 2, 4
Neurobehavioral disorders are increasingly prevalent in children, however their etiology is not well understood. An association between prenatal cellular telephone use and hyperactivity in children has been postulated, yet the direct effects of radiofrequency radiation exposure on neurodevelopment remain unknown. Here we used a mouse model to demonstrate that in-utero radiofrequency exposure from cellular telephones does affect adult behavior. Mice exposed in-utero were hyperactive and had impaired memory as determined using the object recognition, light/dark box and step-down assays. Whole cell patch clamp recordings of miniature excitatory postsynaptic currents (mEPSCs) revealed that these behavioral changes were due to altered neuronal developmental programming. Exposed mice had dose-responsive impaired glutamatergic synaptic transmission onto layer V pyramidal neurons of the prefrontal cortex. We present the first experimental evidence of neuropathology due to in-utero cellular telephone radiation. Further experiments are needed in humans or non-human primates to determine the risk of exposure during pregnancy.
